Pipeline / 01 · Platforms

Multi-Specific Antibody Platform

Tri-specific antibody systems for precision targeting, immune recruitment, and viral escape prevention.

DUAL TRISPECIFIC ANTIBODY PLATFORM Three points of contact, one molecule. HIV-DNS Direct Neutralization Synergy gp120 (Tier-1) gp120 (Tier-2) CD3 effector HIV-IFN Targeted Interferon gp120 Type-I IFN Effector T cell
Dual trispecific antibody platform — HIV-DNS and HIV-IFN constructs designed to achieve durable antiretroviral function through synergistic targeting of viral and host components.
Premise

The problem we are solving.

A conventional antibody sees one target. This is often not enough. Infected cells display multiple markers, tumors mutate to hide single epitopes, and viruses escape single-antibody therapies through predictable evolutionary pressure.

Tri-specific antibodies solve this by binding three targets at once — typically the diseased cell, an immune effector, and a safety checkpoint. The result is a single molecule that identifies the problem, summons the solution, and holds both in productive proximity until the work is done.

Approach

How we tackle it.

MACRO HRD's antibody platform engineers tri-specific constructs de novo, selecting each of the three binding domains for a specific role: recognition of the disease marker, recruitment of a CD4, CD8, or NK effector cell, and prevention of escape through a conserved-epitope anchor.

The resulting molecules are small enough to reach dense tissue environments, stable enough for room-temperature storage, and programmable across indications. A tri-specific backbone validated for one disease can be re-armed with new binding domains within months.

Critically, every construct is engineered against the field's known safety failure modes. Immune-engaging therapies carry well-documented risks — cytokine-release and neurotoxicity among them — and decades of clinical experience have shown that unconstrained immune activation is dangerous. MACRO HRD's designs favor conditional, target-dependent activation, prioritizing a controllable safety profile alongside potency.

PROGRAMME ARCHITECTURE Two engines, complementary mechanisms. HIV-DNS Tier-1 Neutralizing Binds HIV gp120 with high affinity (Kd 10⁻¹² M). Engages CD3 to redirect cytotoxic T cells to neutralize circulating virions and infected cells. Strongly suppresses HIV cell-to-cell transmission. HIV-IFN Targeted Interferon HIV-targeting interferon binds gp120 on all HIV variants. Engages CD3 (TCRα or TCRβ) to redirect strongly-suppressed HIV cell-to-cell transmission. Synergizes with HIV-DNS for multi-mechanism cure.
The MACRO HRD trispecific antibody programme — engineered for HIV functional cure with both directly-neutralizing and immune-engaging mechanisms operating in parallel.
i.
Target triangulation
Computational selection of three epitopes whose combination minimizes escape probability.
ii.
Domain engineering
Phage display and hybridoma generation for high-affinity scFv candidates against each target.
iii.
Backbone assembly
Tri-specific construct assembly with optimized linkers and stability engineering.
iv.
Functional validation
xCELLigence cytotoxicity, immune synapse formation, and escape pressure testing.
Capabilities

What makes this real.

01
Tri-specific targeting
Single molecule, three binding domains — recognition, recruitment, and escape prevention in one reagent.
02
Viral tagging capability
HIV-infected and HPV-transformed cells marked for elimination while sparing healthy tissue.
03
Immune synapse formation
Antibody acts as a bridge between the disease cell and the effector cell, forcing proximity that drives efficacy.
04
Reprogrammable backbone
Validated constructs can be re-engineered for new indications without rebuilding from scratch.
⸻ Continue the platform

“Three points of contact are harder to escape than one. We design antibodies the virus cannot outrun.”